Anti-DDAH2 antibody (C-Term) (STJ70102)

SPECIFICATIONS
ClonalityPolyclonal
HostGoat
ConjugationUnconjugated
IsotypeIgG
STJ70102-100
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General Information

Short DescriptionGoat polyclonal antibody anti-DDAH2 (C-Term) is suitable for use in ELISA, Western Blot, Immunofluorescence and Flow Cytometry research applications.
ApplicationsPep-ELISA/WB/IF/FC
HostGoat
ReactivityHuman/Mouse/Rat/Dog/Cow
NoteSTRICTLY FOR FURTHER SCIENTIFIC RESEARCH USE ONLY (RUO). MUST NOT TO BE USED IN DIAGNOSTIC OR THERAPEUTIC APPLICATIONS.

Product Properties

ClonalityPolyclonal
IsotypeIgG
ConjugationUnconjugated
Concentration0.5 mg/mL
PurificationPurified from goat serum by ammonium sulphate precipitation followed by antigen affinity chromatography using the immunizing peptide.
Dilution RangePeptide ELISA: antibody detection limit dilution 1:16000.
WB: Approx 28kDa band observed in Human Lung lysates (calculated MW of 29.6kDa according to NP_039268.1). Recommended concentration: 0.1-0.3µg/ml. Primary incubation was 1 hour. Preliminar
Formulation0.5 mg/ml in Tris saline, 0.02% sodium azide, pH7.3 with 0.5% bovine serum albumin. NA
Storage InstructionStore at-20°C on receipt and minimise freeze-thaw cycles.

Target Information

Gene SymbolDDAH2
Gene ID23564
Uniprot IDDDAH2_HUMAN
Accession NumberNP_039268.1
Immunogen RegionC-Term
Immunogen SequenceSSLCLVLSTRPHS

Additional Info

Post Translational Modifications Phosphorylated by TBK1. Phosphorylation inhibits the translocation into the mitochondrion upon Sendai viral infection.
Function Putative hydrolase with unknown substrate (Probable). Does not hydrolyze N(G),N(G)-dimethyl-L-arginine (ADMA) which acts as an inhibitor of NOS. In endothelial cells, induces expression of vascular endothelial growth factor (VEGF) via phosphorylation of the transcription factor SP1 by PKA in a process that is independent of NO and NO synthase. Similarly, enhances pancreatic insulin secretion through SP1-mediated transcriptional up-regulation of secretagogin/SCGN, an insulin vesicle docking protein. Upon viral infection, relocates to mitochondria where it promotes mitochondrial fission through activation of DNM1L leading to the inhibition of innate response activation mediated by MAVS.
Protein Name Putative Hydrolase Ddah2
Ddahii
Inactive N(G -N(G-Dimethylarginine Dimethylaminohydrolase 2
Ddah-2
Inactive Dimethylarginine Dimethylaminohydrolase 2
Protein G6a
S-Phase Protein
Cellular Localisation Cytoplasm
Mitochondrion
Translocates From Cytosol To Mitochondrion Upon Il1b Stimulation In Chondrocytes
(Microbial Infection) Translocates To The Mitochondrion Upon Sendai Viral Infection
Alternative Antibody Names Anti-Putative Hydrolase Ddah2 antibody
Anti-Ddahii antibody
Anti-Inactive N(G -N(G-Dimethylarginine Dimethylaminohydrolase 2 antibody
Anti-Ddah-2 antibody
Anti-Inactive Dimethylarginine Dimethylaminohydrolase 2 antibody
Anti-Protein G6a antibody
Anti-S-Phase Protein antibody
Anti-DDAH2 antibody
Anti-DDAH antibody
Anti-G6A antibody
Anti-NG30 antibody

Information sourced from Uniprot.org

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