Anti-BNIP3L antibody (Internal Region) (STJ13100734)

SPECIFICATIONS
ClonalityPolyclonal
HostRabbit
ConjugationUnconjugated
IsotypeIgG
ImmunogenA synthetic peptide from the internal region of rat BNIP3L (BCL2/adenovirus E1B 19 kDa protein-interacting protein 3-like) conjugated to an immunogenic carrier protein was used as the antigen. The peptide is homologous in human and mouse.
STJ13100734-100
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General Information

Short DescriptionRabbit polyclonal anti-BNIP3L (Internal Region) for use in IHC and WB in Human, Rat and Mouse samples. Datasheet included with dilution recommendations, and related reagents.
ApplicationsIHC/WB
HostRabbit
ReactivityHuman/Rat/Mouse
NoteSTRICTLY FOR FURTHER SCIENTIFIC RESEARCH USE ONLY (RUO). MUST NOT TO BE USED IN DIAGNOSTIC OR THERAPEUTIC APPLICATIONS.

Product Properties

ClonalityPolyclonal
IsotypeIgG
ConjugationUnconjugated
PurificationIgG purified
Dilution RangeIHC, WB. A dilution of 1:300 to 1:2000 is recommended. The optimal dilution should be determined by the end user. Not yet tested in other applications.
FormulationLyophilised
Storage InstructionMaintain the lyophilised/reconstituted antibodies frozen at-20°C for long term storage and refrigerated at 2-8°C for a shorter term. When reconstituting, Glycerol (1:1) may be added for an additional stability. Avoid freeze and thaw cycles.

Target Information

Gene SymbolBNIP3L
Gene ID665
Uniprot IDBNI3L_HUMAN
ImmunogenA synthetic peptide from the internal region of rat BNIP3L (BCL2/adenovirus E1B 19 kDa protein-interacting protein 3-like) conjugated to an immunogenic carrier protein was used as the antigen. The peptide is homologous in human and mouse.
Immunogen RegionInternal Region
SpecificitySpecific for BNIP3L.

Additional Info

Function Induces apoptosis. Interacts with viral and cellular anti-apoptosis proteins. Can overcome the suppressors BCL-2 and BCL-XL, although high levels of BCL-XL expression will inhibit apoptosis. Inhibits apoptosis induced by BNIP3. Involved in mitochondrial quality control via its interaction with SPATA18/MIEAP: in response to mitochondrial damage, participates in mitochondrial protein catabolic process (also named MALM) leading to the degradation of damaged proteins inside mitochondria. The physical interaction of SPATA18/MIEAP, BNIP3 and BNIP3L/NIX at the mitochondrial outer membrane regulates the opening of a pore in the mitochondrial double membrane in order to mediate the translocation of lysosomal proteins from the cytoplasm to the mitochondrial matrix. May function as a tumor suppressor.
Protein Name Bcl2/Adenovirus E1b 19 Kda Protein-Interacting Protein 3-Like
Adenovirus E1b19k-Binding Protein B5
Bcl2/Adenovirus E1b 19 Kda Protein-Interacting Protein 3a
Nip3-Like Protein X
Nip3l
Database Links Reactome: R-HSA-6803204
Cellular Localisation Nucleus Envelope
Endoplasmic Reticulum
Mitochondrion Outer Membrane
Membrane
Single-Pass Membrane Protein
Colocalizes With Spata18 At The Mitochondrion Outer Membrane
Alternative Antibody Names Anti-Bcl2/Adenovirus E1b 19 Kda Protein-Interacting Protein 3-Like antibody
Anti-Adenovirus E1b19k-Binding Protein B5 antibody
Anti-Bcl2/Adenovirus E1b 19 Kda Protein-Interacting Protein 3a antibody
Anti-Nip3-Like Protein X antibody
Anti-Nip3l antibody
Anti-BNIP3L antibody
Anti-BNIP3A antibody
Anti-BNIP3H antibody
Anti-NIX antibody

Information sourced from Uniprot.org

Citations

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