Human IFNAR1 protein (Recombinant-Active) (C-His) (STJP011894)

SPECIFICATIONS
HostMammalian Cells
ConjugationUnconjugated
ImmunogenHomo sapiens (Human)
STJP011894-100
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General Information

Short DescriptionRecombinant-Active-Human IFNAR1-C-His protein was developed from mammalian cells and has a target region of C-His. For use in research applications.
ApplicationsELISA/WB
HostMammalian Cells
NoteSTRICTLY FOR FURTHER SCIENTIFIC RESEARCH USE ONLY (RUO). MUST NOT BE USED IN DIAGNOSTIC OR THERAPEUTIC APPLICATIONS.

Product Properties

ConjugationUnconjugated
Dilution RangeReconstitute in sterile water for a stock solution.
FormulationLyophilized from a solution in PBS pH 7.4, 5% Trehalose, 5% Mannitol.
Storage InstructionUse a manual defrost freezer and avoid repeated freeze thaw cycles. Store at 2 to 8°C for one week. Store at-20 to-80°C for twelve months from the date of receipt.
Endotoxin<0.1 EU/Mu g of the protein by the LAL method.
ImmunoreactivityMeasured by its binding ability in a functional ELISA. When recombinant IFNAR1 is present at 10 Mu g/mL, it binds to recombinant human IFNAR2 in the presence of recombinant human IFN alpha 2. The concentration of recombinant human IFNAR2 that produce

Target Information

Gene SymbolIFNAR1
Gene ID3454
Uniprot IDINAR1_HUMAN
ImmunogenHomo sapiens (Human)
Immunogen RegionLys28-Lys436

Additional Info

Post Translational Modifications Ubiquitinated, leading to its internalization and degradation. Polyubiquitinated via 'Lys-48'-linked and 'Lys-63'-linked ubiquitin chains, leading to receptor internalization and lysosomal degradation. The 'Lys-63'-linked ubiquitin chains are cleaved off by the BRISC complex. Phosphorylated on tyrosine residues in response to interferon-binding: phosphorylation by TYK2 tyrosine kinase creates docking sites for STAT proteins. Phosphorylated on serine residues in response to interferon binding.this promotes interaction with FBXW11 and ubiquitination. Palmitoylation at Cys-463 is required for the activation of STAT1 and STAT2.
Function Together with IFNAR2, forms the heterodimeric receptor for type I interferons (including interferons alpha, beta, epsilon, omega and kappa). Type I interferon binding activates the JAK-STAT signaling cascade, resulting in transcriptional activation or repression of interferon-regulated genes that encode the effectors of the interferon response. Mechanistically, type I interferon-binding brings the IFNAR1 and IFNAR2 subunits into close proximity with one another, driving their associated Janus kinases (JAKs) (TYK2 bound to IFNAR1 and JAK1 bound to IFNAR2) to cross-phosphorylate one another. The activated kinases phosphorylate specific tyrosine residues on the intracellular domains of IFNAR1 and IFNAR2, forming docking sites for the STAT transcription factors. STAT proteins are then phosphorylated by the JAKs, promoting their translocation into the nucleus to regulate expression of interferon-regulated genes. Can also act independently of IFNAR2: form an active IFNB1 receptor by itself and activate a signaling cascade that does not involve activation of the JAK-STAT pathway.
Protein Name Interferon Alpha/Beta Receptor 1
Ifn-R-1
Ifn-Alpha/Beta Receptor 1
Cytokine Receptor Class-Ii Member 1
Cytokine Receptor Family 2 Member 1
Crf2-1
Type I Interferon Receptor 1
Database Links Reactome: R-HSA-909733
Reactome: R-HSA-912694
Reactome: R-HSA-9679191
Reactome: R-HSA-9705671
Reactome: R-HSA-9833109
Cellular Localisation Isoform 1: Cell Membrane
Single-Pass Type I Membrane Protein
Late Endosome
Lysosome
Interferon Binding Triggers Internalization Of The Receptor From The Cell Membrane Into Endosomes And Then Into Lysosomes
Alternative Protein Names Interferon Alpha/Beta Receptor 1 protein
Ifn-R-1 protein
Ifn-Alpha/Beta Receptor 1 protein
Cytokine Receptor Class-Ii Member 1 protein
Cytokine Receptor Family 2 Member 1 protein
Crf2-1 protein
Type I Interferon Receptor 1 protein
IFNAR1 protein
IFNAR protein

Information sourced from Uniprot.org

Citations

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