Anti-Pin1 antibody (118-168) {Biotin} (STJ502497)

SPECIFICATIONS
ClonalityPolyclonal
HostRabbit
ConjugationBiotin
IsotypeIgG
ImmunogenSynthetic peptide taken within amino acid regionΒ 118-168 on human peptidyl-prolyl cis-trans isomerase NIMA-interacting 1 protein.
STJ502497-100
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General Information

Short DescriptionRabbit polyclonal antibody anti-Pin1 (118-168) is suitable for use in ELISA, Immunoprecipitation and Western Blot research applications.
ApplicationsELISA/IP/WB
HostRabbit
ReactivityHuman/Monkey/Mouse/Rat
NoteSTRICTLY FOR FURTHER SCIENTIFIC RESEARCH USE ONLY (RUO). MUST NOT TO BE USED IN DIAGNOSTIC OR THERAPEUTIC APPLICATIONS.

Product Properties

ClonalityPolyclonal
IsotypeIgG
ConjugationBiotin
Concentration0.56-0.62 Β΅g/Β΅l
PurificationAffinity Purified
Dilution RangeWB: 1:500
DB: 1:10, 000
ELISA: 1:10, 000
IP: 1:200
FormulationContains Tris, HCl/Glycine buffer pH 7.4-7.8, 30% Glycerol and 0.5% BSA, along with cryo-protective agents, Hepes, and long-term preservatives (0.02% Sodium Azide).
Storage InstructionStore at-20Β°C for long term storage. Avoid freeze-thaw cycles.

Target Information

ImmunogenSynthetic peptide taken within amino acid regionΒ 118-168 on human peptidyl-prolyl cis-trans isomerase NIMA-interacting 1 protein.
Immunogen Region118-168

Additional Info

Background Peptidyl-prolyl cis/trans isomerase (PPIase) that binds to and isomerizes specific phosphorylated Ser/Thr-Pro (pSer/Thr-Pro) motifs in a subset of proteins, resulting in conformational changes in the proteins (PubMed:21497122, PubMed:22033920). Displays a preference for an acidic residue N-terminal to the isomerized proline bond. Regulates mitosis presumably by interacting with NIMA and attenuating its mitosis-promoting activity. Down-regulates kinase activity of BTK (PubMed:16644721). Can transactivate multiple oncogenes and induce centrosome amplification, chromosome instability and cell transformation. Required for the efficient dephosphorylation and recycling of RAF1 after mitogen activation (PubMed:15664191). Binds and targets PML and BCL6 for degradation in a phosphorylation-dependent manner (PubMed:17828269). Acts as a regulator of JNK cascade by binding to phosphorylated FBXW7, disrupting FBXW7 dimerization and promoting FBXW7 autoubiquitination and degradation: degradation of FBXW7 leads to subsequent stabilization of JUN (PubMed:22608923).

Information sourced from Uniprot.org

Citations

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