Anti-Phospho-NOS3-Thr494 antibody (462-511 aa) (STJ90356)

SPECIFICATIONS
ClonalityPolyclonal
HostRabbit
ConjugationUnconjugated
IsotypeIgG
ImmunogenThe antiserum was produced against synthesized peptide derived from the human eNOS around the phosphorylation site of Thr494 at the amino acid range 462-511
STJ90356
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General Information

Short DescriptionRabbit polyclonal anti-Phospho-Nitric oxide synthase 3-Thr494 (462-511 aa) for use in WB and ELISA in Human, Mouse and Rat samples. Datasheet included with dilution recommendations, and related reagents.
ApplicationsWB/ELISA
HostRabbit
ReactivityHuman/Mouse/Rat
NoteSTRICTLY FOR FURTHER SCIENTIFIC RESEARCH USE ONLY (RUO). MUST NOT TO BE USED IN DIAGNOSTIC OR THERAPEUTIC APPLICATIONS.

Product Properties

ClonalityPolyclonal
IsotypeIgG
ConjugationUnconjugated
Concentration1 mg/mL
PurificationThe antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen.
Dilution RangeWB 1:500-1:2000
ELISA 1:5000
FormulationLiquid in PBS containing 50% Glycerol, 0.5% BSA and 0.02% Sodium Azide.
Storage InstructionStore at-20ยฐC for up to 1 year from the date of receipt, and avoid repeat freeze-thaw cycles.

Target Information

Gene SymbolNOS3
Gene ID4846
Uniprot IDNOS3_HUMAN
ImmunogenThe antiserum was produced against synthesized peptide derived from the human eNOS around the phosphorylation site of Thr494 at the amino acid range 462-511
Immunogen Region462-511 aa
SpecificityPhospho-NOS3 (T494) Polyclonal Antibody detects endogenous levels of NOS3 protein only when phosphorylated at T494.

Additional Info

Post Translational Modifications Phosphorylation by AMPK at Ser-1177 in the presence of Ca(2+)-calmodulin (CaM) activates activity. In absence of Ca(2+)-calmodulin, AMPK also phosphorylates Thr-495, resulting in inhibition of activity. Phosphorylation of Ser-114 by CDK5 reduces activity.
Function Produces nitric oxide (NO) which is implicated in vascular smooth muscle relaxation through a cGMP-mediated signal transduction pathway. NO mediates vascular endothelial growth factor (VEGF)-induced angiogenesis in coronary vessels and promotes blood clotting through the activation of platelets. Isoform eNOS13C: Lacks eNOS activity, dominant-negative form that may down-regulate eNOS activity by forming heterodimers with isoform 1.
Protein Name Nitric Oxide Synthase 3
Constitutive Nos
Cnos
Ec-Nos
Nos Type Iii
Nosiii
Nitric Oxide Synthase - Endothelial
Endothelial Nos
Enos
Database Links Reactome: R-HSA-1222556
Reactome: R-HSA-1474151
Reactome: R-HSA-203615
Reactome: R-HSA-203641
Reactome: R-HSA-203754
Reactome: R-HSA-392154
Reactome: R-HSA-5218920
Reactome: R-HSA-9009391
Reactome: R-HSA-9856530
Cellular Localisation Cell Membrane
Membrane
Caveola
Cytoplasm
Cytoskeleton
Golgi Apparatus
Specifically Associates With Actin Cytoskeleton In The G2 Phase Of The Cell Cycle
Which Is Favored By Interaction With Nosip And Results In A Reduced Enzymatic Activity
Alternative Antibody Names Anti-Nitric Oxide Synthase 3 antibody
Anti-Constitutive Nos antibody
Anti-Cnos antibody
Anti-Ec-Nos antibody
Anti-Nos Type Iii antibody
Anti-Nosiii antibody
Anti-Nitric Oxide Synthase - Endothelial antibody
Anti-Endothelial Nos antibody
Anti-Enos antibody
Anti-NOS3 antibody

Information sourced from Uniprot.org

Citations

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